![]() Arterial blood ( breathing ambient air) was collected while exhaled O2 and CO2 concentrations were measured, yielding alveolar-arterial differences for each gas (AaPO2, aAPCO2) from which shunt and alveolar dead space were computed. METHODS: We studied 30 patients (22 males, age 49.9☑3.5â years) 3-15â days from symptom onset and again during recovery, 55☑0â days later (n=17). We aimed to measure shunt and alveolar deadspace in moderate COVID-19 during acute illness and recovery. We propose that measuring respiratory gas exchange enables detection and quantification of these abnormalities. ABSTRACT BACKGROUND: Pathological evidence suggests that COVID-19 pulmonary infection involves both alveolar damage (causing shunt) and diffuse micro-vascular thrombus formation (causing alveolar dead space).
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